TEAD1

Information TEAD1

Description

This gene encodes a ubiquitous transcriptional enhancer factor that is a member of the TEA/ATTS domain family. This protein directs the transactivation of a wide variety of genes and, in placental cells, also acts as a transcriptional repressor. Mutations in this gene cause Sveinsson's chorioretinal atrophy. Additional transcript variants have been described but their full-length natures have not been experimentally verified. [provided by RefSeq, May 2010]

Full Name

TEA domain transcription factor 1

Source NCBI

ReMap Statistics

Datasets
9
Biotypes
9
Peaks
423,641
Non-redundant peaks
272,665

TF Classification

Super Class
Helix-turn-helix domains
Class
TEA domain factors
Familly
TEF-1-related factors
Sub Familly
None

Source TFClass

External IDs

JASPAR
MA0090
Ensembl
ENSG00000187079
UniProt
P28347
Genevisible
P28347
RefSeq
NM_021961
Aliases
AA; NTEF-1; REF1; TCF-13; TCF13; TEAD-1; TEF-1
All peaks TEAD1
Download BED file
Non redundant peaks TEAD1
Download BED file
SEQUENCES TEAD1
Download FASTA file
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Datasets Table for TEAD1

Target name Target modification Ecotype/Strain Biotype Biotype modification Source Species Experiment Peaks
TEAD1 pancreas 12D ENA Homo sapiens ERP008682 22,965
TEAD1 Hep-G2 ENCODE Homo sapiens ENCSR497JLX 100,070
TEAD1 MCF-7 GEO Homo sapiens GSE107013 93,503
TEAD1 T-47D GEO Homo sapiens GSE107013 81,464
TEAD1 CCLP1 GEO Homo sapiens GSE62272 7,669
TEAD1 HFOB DIFF GEO Homo sapiens GSE82295 1,100
TEAD1 HUCCT1 GEO Homo sapiens GSE68296 52,281
TEAD1 MSTO GEO Homo sapiens GSE68170 29,852
TEAD1 H69 GEO Homo sapiens GSE62274 34,737
Target name Target modification Ecotype/Strain Biotype Biotype modification Source Species Experiment Peaks

ReMap is a database of transcriptional regulators peaks derived from curated ChIP-seq, ChIP-exo, DAP-seq experiments in Human and Thaliana.

You are using the 2020 ReMap (3rd) release.
The ReMap catalogues (2020, 2018, 2015) are under CC BY-NC 4.0 international license, while ReMapEnrich, remap-pipeline under GNU GPLv3 licence.

Inserm TAGC
AMU AMU-MESO

This work was granted access to the HPC resources of Aix-Marseille Université financed by the project Equip@Meso (ANR-10-EQPX-29-01) of the program "Investissements d’Avenir" supervised by the Agence Nationale de la Recherche.